A2 Negative feedback - Blood Glucose?

Hi, we haven't really covered this section (teacher pulled a "now i want you to go away and learn this" on us -.-)
I'm going to summarise what i assume is correct for this section and want people to tell me how right/wrong i am. Ty

When there's high blood glucose, the molecules bind to receptors the beta cell membranes in Islets of Langerhans. The cells secrete insulin into the blood, which binds to receptors on liver cells. The insulin causes the permeability of the cells to increase so they take up more glucose. The glucose is converted into glycogen which is stored, hence lowering glucose levels.
When there's low blood glucose, cells in the pancreas secrete glucagon into the bloodstream, which binds to receptors on the liver cells. This converts the stored glycogen into glucose, increasing glucose concentration.

Is this ^ right? That's the basic gist that i've got from reading about it, correct me if i'm wrong please.

I've done a bit about thermoregulation in humans, and imo it's pretty well the same for animals?
I'll add osmoregulation too, thanks.

Hi, we haven't really covered this section (teacher pulled a "now i want you to go away and learn this" on us -.-)
I'm going to summarise what i assume is correct for this section and want people to tell me how right/wrong i am. Ty

When there's high blood glucose, the molecules bind to receptors the beta cell membranes in Islets of Langerhans. The cells secrete insulin into the blood, which binds to receptors on liver cells. The insulin causes the permeability of the cells to increase so they take up more glucose. The glucose is converted into glycogen which is stored, hence lowering glucose levels.
When there's low blood glucose, cells in the pancreas secrete glucagon into the bloodstream, which binds to receptors on the liver cells. This converts the stored glycogen into glucose, increasing glucose concentration.

Is this ^ right? That's the basic gist that i've got from reading about it, correct me if i'm wrong please.

@ the bit in bold; NO! it is sufficient to know that decreased blood glucose causes the beta cells to secrete insulin but that is not the (main and best characterised) mechanism by which they do so.

also note; other peripheral tissues can store glycogen (e.g. muscle glycogen > liver glycogen) and it is not the only way to convert glucose to a storage molecule (!!). in addition; other tissues respond to insulin (adipose etc) whereas glycogen's effects are primarily at the liver. the mechanisms by which insulin and glucagon influence cellular metabolism are also more complex that simply turning on/off the production of glycogen. for example; do you want glycolysis going on if you have low blood glucose (where there is increased glucagon)?

just so you don't go away thinking about glucose metabolism the wrong way - it's far far more complex and finely regulated than you are suggesting .

ps: for a fun extra thought, how might you suppose 'glucose permeability' in the liver (and other tissues) is increased by the action of insulin? recalling from AS the action of a certain kind of membrane protein

Pardon?