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    (Original post by theambitiousguru)
    Passive immunity Q
    does this make sense then?
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    Who else thinks the grade boundaries will be very high?
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    (Original post by theambitiousguru)
    Yes and I also out about when infected it would be a primary response which is slower and therefore would cause harm / symptoms
    kk good
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    (Original post by Kira Yagami)
    does this make sense then?
    Yes as the blood will contain memory cells and the specific antibodies needed to kill the pathogen
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    did anyone else put thicker alveolar walls so increased diffusion pathway and lung tissue loses elasticity so cannot recoil for the qs on what happens to lungs after smoking?
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    What does everyone think the grade boundaries will be like


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    (Original post by Anam)
    did anyone else put thicker alveolar walls so increased diffusion pathway and lung tissue loses elasticity so cannot recoil for the qs on what happens to lungs after smoking?
    Because it was to do with smoking - linked to emphysema

    Alveoli burst - Dec SA
    Elastin becomes permanently stretched - CG not maintained - struggle to breathe out
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    (Original post by Anam)
    did anyone else put thicker alveolar walls so increased diffusion pathway and lung tissue loses elasticity so cannot recoil for the qs on what happens to lungs after smoking?
    Exactly what I put, increased diffusion pathway and lower surface area
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    About the decrease in ph when Triglecrides are hydloyseD:

    forms an acidic solution?

    and the one about why ph remains constant at 2

    because Trigly not hydloysed as enzyme is limiting as enzymes active site are saturated/occupied


    (Mind the spelling, I'm typing quickly)
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    (Original post by theambitiousguru)
    Who else thinks the grade boundaries will be very high?
    Me, probably 49 for an A
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    (Original post by Kira Yagami)
    About the decrease in ph when Triglecrides are hydloyseD:

    forms an acidic solution?

    and the one about why ph remains constant at 2

    because Trigly not hydloysed as enzyme is limiting as enzymes active site are saturated/occupied


    (Mind the spelling, I'm typing quickly)
    I said triglycerides were limiting factor because it didn't specify enzyme concentration

    Lactase hydrolyses triglycerides into fatty acids, so oh lowers
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    What did you all put for the conclusions for the untreated lactose and treated lactose?
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    (Original post by Bennyab)
    Exactly what I put, increased diffusion pathway and lower surface area
    Don't think you needed to explain. It was just desribe to features or something.

    But yeah I still explained just incase.
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    (Original post by Kira Yagami)
    About the decrease in ph when Triglecrides are hydloyseD:

    forms an acidic solution?

    and the one about why ph remains constant at 2

    because Trigly not hydloysed as enzyme is limiting as enzymes active site are saturated/occupied


    (Mind the spelling, I'm typing quickly)
    I put becomes more acidic as ester bond broken therefore forms carboxlyic acid which is acidic

    Second Q I put all ester bonds had been broken and all triglycerides had been hydrolysed
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    i found the paper harder than last years, what i found hard seems to be what everybody else is finding hard here so hopefully the grade boundaries will be low i hope it will be like 41-43 for an A
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    (Original post by gcsekid)
    I could do but I have answers written on it
    If u still want it I can send u it
    Is this the AQA AS or A level biology exam? If it's the AS could I see it too please?
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    thought that was quite a nice exam tbh, grade boundaries will be really high especially since it's only a2 students sitting it
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    Can someone make a full unofficial MS please?
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    What else did people put for the triglyceride question about ph constant?

    Were triglycerides limiting factor (what I put) or enzyme?
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    Was there a question about where trypsin was made in the pancreas?
 
 
 
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