TSR Med Students&#x27; Society Part III

Original post by Fission_Mailed

I quite like my surname, it doesn't sound particularly unusual, IMO, but I've never met anyone else (who I'm not related to) that shares it.

Ditto

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Fission_Mailed

Original post by Kinkerz

Ditto

I'd laugh if we had the same name.

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ThisLittlePiggy

12

Anyone managed to log onto NHS.net using a mac? How'd you manage it?

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Mushi_master

17

Original post by billet-doux

Agreed! They are such a mission :frown:

How is 3rd year going?! :biggrin:

It's pretty ace all in all tbh. Being in Chichester for my first rotation is definitely a good thing, very social atmosphere and everyone's so willing to give us their time and to teach us. Enjoying gastro far more than I thought I would too.

Must say it's a lot of work, probably more to do than in second year but it's a much better kind of busy actually seeing patients and doing practical things. Way, way better than pre-clinical stuff! Second year was such a chore (not very reassuring I know!).

Reply 6665

Original post by Mushi_master

It's pretty ace all in all tbh. Being in Chichester for my first rotation is definitely a good thing, very social atmosphere and everyone's so willing to give us their time and to teach us. Enjoying gastro far more than I thought I would too.

Must say it's a lot of work, probably more to do than in second year but it's a much better kind of busy actually seeing patients and doing practical things. Way, way better than pre-clinical stuff! Second year was such a chore (not very reassuring I know!).

Chinchester? Christ, isn't that a treck?

Reply 6666

So, I'm writing up a PBL for blood transfusions which I always thought I understood.

However, I was just writing out about O- being the universal doner but I kind of don't understand it anymore. I understand it has no antigens on the surface of the RBCs to be caught by the recipients antibodies but surely it has A and B antibodies in it? So wouldn't that cause a reaction between the recipients A/B/AB antigens and the donors antibodies? I know rhesus it won't because *most* people don't have rhesus antibodies unless they've been previously exposed to it.

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Becca-Sarah

19

Original post by Helenia

Given the **** I got at school for my surname, I have no idea why anyone would pick it voluntarily.

I would happily trade :sigh:

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Carpediemxx

17

Passed my first finals long case :biggrin:

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Jessaay!

Original post by RollerBall

So, I'm writing up a PBL for blood transfusions which I always thought I understood.

However, I was just writing out about O- being the universal doner but I kind of don't understand it anymore. I understand it has no antigens on the surface of the RBCs to be caught by the recipients antibodies but surely it has A and B antibodies in it? So wouldn't that cause a reaction between the recipients A/B/AB antigens and the donors antibodies? I know rhesus it won't because *most* people don't have rhesus antibodies unless they've been previously exposed to it.

As far as I was told, the reason it's universal is because it's universal for a red cell transfusion. I.e. they transfer the red cells, not the serum/other cells in the donor blood. Therefore the antibodies won't be transfused. There are different types of transfusion which transfer different aspects of the blood and I believe you can make a full blood transfusion, so serum, red cells etc. Someone correct me if I'm wrong, but that's what I was made to believe.

Reply 6670

Original post by Jessaay!

As far as I was told, the reason it's universal is because it's universal for a red cell transfusion. I.e. they transfer the red cells, not the serum/other cells in the donor blood. Therefore the antibodies won't be transfused. There are different types of transfusion which transfer different aspects of the blood and I believe you can make a full blood transfusion, so serum, red cells etc. Someone correct me if I'm wrong, but that's what I was made to believe.

Yeah, I think you're right. I had a look around after I posted and there are seperate plasma (contains antibodies but not antigens), platlets (contain neither but should try to match for remnent red blood cells) and RBC transfusions (follow the standard rules).

From what I have gathered they no longer do full blood transfusions which is why it is not neccessary to match antibodies as well as antigens.

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GodspeedGehenna

3

Original post by Jessaay!

As far as I was told, the reason it's universal is because it's universal for a red cell transfusion. I.e. they transfer the red cells, not the serum/other cells in the donor blood. Therefore the antibodies won't be transfused. There are different types of transfusion which transfer different aspects of the blood and I believe you can make a full blood transfusion, so serum, red cells etc. Someone correct me if I'm wrong, but that's what I was made to believe.

Sounds legit.

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Lantana

10

Original post by RollerBall

So, I'm writing up a PBL for blood transfusions which I always thought I understood.

However, I was just writing out about O- being the universal doner but I kind of don't understand it anymore. I understand it has no antigens on the surface of the RBCs to be caught by the recipients antibodies but surely it has A and B antibodies in it? So wouldn't that cause a reaction between the recipients A/B/AB antigens and the donors antibodies? I know rhesus it won't because *most* people don't have rhesus antibodies unless they've been previously exposed to it.

As above, but also you want to think about volumes. There will be a tiny amount of a/b graft antibodies against lots of a/b/ab host cells whereas it's much more problematic if there are lots of host antibodies against the graft cells. However, there is graft vs. host disease :wink:

Reply 6673

Two PBLs done. Can't be bothered with thalassemia. I shall crack on tomorrow and try to get that one done and the four Metabolism PBLs I'm behind on. Then rock on the lecture write ups.

This work melarky is far too much work.

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Original post by Lantana

As above, but also you want to think about volumes. There will be a tiny amount of a/b graft antibodies against lots of a/b/ab host cells whereas it's much more problematic if there are lots of host antibodies against the graft cells. However, there is graft vs. host disease :wink:

Isn't GvH something completely different? As in it's from marrow transplants and the creation of antibodies against the hosts cells from the graft marrow?

Reply 6675

billet-doux

14

Original post by Mushi_master

It's pretty ace all in all tbh. Being in Chichester for my first rotation is definitely a good thing, very social atmosphere and everyone's so willing to give us their time and to teach us. Enjoying gastro far more than I thought I would too.

Must say it's a lot of work, probably more to do than in second year but it's a much better kind of busy actually seeing patients and doing practical things. Way, way better than pre-clinical stuff! Second year was such a chore (not very reassuring I know!).

That sounds pretty ace! Did you choose to be placed outside of London or is it all pretty random? When I hear stories like yours in comparison to the horror stories of iBSc, it makes me pretty tempted to just go straight into clinics :tongue:

Yes, second year is such an effort. It's getting really tedious and we're not even half way through!

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Philosoraptor

18

Had a final year GKT student at our GP - genuinely impressed guys well done - I'll take the mick slightly less frequently from now on :p:

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Blatant Troll