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    (Original post by haydyb123)
    Read topic 7's spread on Immune system, those macrophages will be right on it
    The usual boring stuff like fruit/veg/hot drinks....
    Vix vapour rub?!

    Seriously hope you feel better for tomorrow...
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    (Original post by Jennie_pro)
    OK I think it's safe to say i would have got no marks for that question!!haha
    I know, me neither!! well this is looking great for tomorrow
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    I wouldn't take this as gospel, see the mark scheme previous post!!!
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    Hate those sort of questions. Think i was kinda right referring to the oxygen debt and FAD and NAD business, but god, even reading the answer and I'm little the wiser!
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    (Original post by haydyb123)
    Read topic 7's spread on Immune system, those macrophages will be right on it
    i know but i want to treat my symptoms so im alert for this exam
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    is ubiquitin ligase a signal protein that inities the ubiquitin proteasome pathway causing atrophy? could anybody explain the relationship between all this stuff with gene expression, it would be greatly appreciated
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    Could someone please send me the mark scheme for the Jan 2011 paper?

    Many thanks'!

    JP
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    this is jan 2011 mark scheme.......
    hope u all do well 2moro, I kno I wuldn't
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    (Original post by Jennie_pro)
    Could someone please send me the mark scheme for the Jan 2011 paper?

    Many thanks'!

    JP

    here u go
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  2. File Type: pdf 6BI05_01_rms_20110309.pdf (132.3 KB, 65 views)
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    Hiiii! i'm sorry guys but i am really struggling with nerves/synapses etc and respiration. For some reason i can't get my head around any of it! would anyone be able to give me a quick summary of important/key points from their notes etc? love you forever if you doooooooooooooooo! <3
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    (Original post by guichiguichi)
    is ubiquitin ligase a signal protein that inities the ubiquitin proteasome pathway causing atrophy? could anybody explain the relationship between all this stuff with gene expression, it would be greatly appreciated
    use this for the article and ull be fine

    54265374-Questions-and-Answers-for-Scientific-Article-June-2011.pdf
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    (Original post by sar_91)
    this is jan 2011 mark scheme.......
    hope u all do well 2moro, I kno I wuldn't
    Many thanks! Good luck to you too

    I think it is going to be very hard compared to the other exams!
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    (Original post by abuelzouz)
    hey
    u got 2 types of phytocromes
    red and far red
    Pr and Pfr
    Pr absorbs red light and change form to Pfr
    while Pfr absorbs far red light and change form to Pr
    Pr stimulate flowering in Long Day Plant
    inhibits flowering in Short Day Plant
    Pfr stimulate flowering in Short Day Plant
    inhibit flowering in Long Day Plant
    First you need to be aware of the reaction that takes place

    RED LIGHT - Day
    FAR RED LIGHT - Night
    So in red light/DAY Pr is changed to Pfr
    and in far red light/NIGHT Pfr is is changed to Pr

    You need to be aware of two types of plants:
    • LDP (long day plants)
    • SDP (short day plants)

    LDP can be referred to as SNP 'short night plants' for better understanding but do not call them this during the exam!
    • Pfr STIMULATES flowering
    • Requires MAXIMUM hours of darkness, any longer and Pfr will be changed into Pr so no flowering.

    SDP 'Long night plants'
    • Pfr INHIBITS flowering
    • requires MINIMUM length of darkness - requires enough far red light to change sufficient amount of Pfr to remove the inhibition
    Remember its the length of DARKNESS that determines whether a plant flowers. This can be demonstrated by:
    Placing a SDP 'long night plant' and exposing it to say 3 hours of day and 9 hours of darkness. The result is flowering. However if you distrub this single period of darkness by flashing red light (day - changes Pr to Pfr) there is NO flowering.
    Therefore the conclusion is that the period of darkness is the CRITICAL FACTOR in inducing flowering.
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    (Original post by amnah_70)
    use this for the article and ull be fine

    54265374-Questions-and-Answers-for-Scientific-Article-June-2011.pdf
    Good lord this is extensive, many many thanks!
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    Hi does anyone have the jan 2010 paper and mark scheme? It would be greatly appreciated
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    (Original post by Choca Mocha)
    Hi does anyone have the jan 2010 paper and mark scheme? It would be greatly appreciated
    the new paper started from june 2010 onwards, no paper for jan 2010
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    (Original post by amnah_70)
    use this for the article and ull be fine

    54265374-Questions-and-Answers-for-Scientific-Article-June-2011.pdf
    thankyou so much!!! you are a lifesaver
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    (Original post by Maria1234)
    due a mutation his epo receptors did not shut down, which was a good mutation for him!becuase normally when oxgen levels are normal the receptor will shut down the epo production , I think his EPO receptrs remained open all the aim hence the crucial feedback was not truned on, when the level of oxgyen was high or low!
    I thought the normal EPO receptor was responsible for shutting down EPO production once Oxygen levels become normal. So wouldn't his mutation have caused the receptor to not be formed/incorrectly formed so EPO production continues past normal oxygen levels?
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    Shouldn't we all put up and compare our "bullet points" for the article?

    Should help to get a clearer understanding ...
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    "How do auxins lead to cell elongation?"

    For this question, would you talk about auxins binding to receptors on cells in the zone of elongation causing a secondary messenger that brings about changes in gene expression, or would you talk about the auxin indirectly causing the acidification of the cell wall and the process that occurs afterwards?
 
 
 

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