Competition - post a photo you've taken of nature >>

Edexcel A2 Biology Unit 5 (6BIO5) - 22/06/2011- OFFICIAL THREAD !

Scroll to see replies

Reply 2480

Lynam

I don't understand aerobic respiration atall, is there a chance it won't come up ?

Reply 2481

k256_93

Original post by haydyb123

No worries, I don't suppose you have any notes on the core practicals do you? :smile:

Ah.. really sorry mate but don't have anything on the computer.

Reply 2482

shanshine

can anyone post a link to the examinor reports/grade boundries for the previous 2 papers?

Reply 2483

Parthenon93

Can anyone please explain what is the QT period and what exactly is "long QT" syndrome and how it "can lead to sudden death".

Reply 2484

StudentToday

outline how gene expression may play a part is the day-by-day regulation of conditions in the body?? (5)
Does any1 know the answer to this??? its about homeostasis but the gene expression has thrown me lool.
Please any suggestions its bugging me!!

Charli :smile:

Reply 2485

claire4095

How are you all revising the scientific article?
Will just reading it over a few times be enough? :/

Reply 2486

Jammers123

Original post by Lynam

I don't understand aerobic respiration atall, is there a chance it won't come up ?

Highly unlikely that it won't come up, there's a massive section on it!!!

Reply 2487

Jammers123

Original post by Parthenon93

Can anyone please explain what is the QT period and what exactly is "long QT" syndrome and how it "can lead to sudden death".

I would have thought QT is the length of time taken for ventricular systole :wink:

Reply 2488

Parthenon93

Original post by StudentToday

Could be along the lines that ... regular use of muscles for normal activities and exercise causes micro tears in muscles ... stress and exercise triggers transcription of IFG-1 gene (may be by affect of stress hormones) ... IFG-1 adds more proteins to muscle to repair tears, muscle mass increaes ... more exercise ... more tear ... more IFG-1 :eek:

Or you can just outline transcription of proteins which neurotransmitters ...

Weird question ... :eek:

Reply 2489

Lynam

Original post by Jammers123

Highly unlikely that it won't come up, there's a massive section on it!!!

Ergh i know but a lot of the stuff came up in january, i understand everything else just all the chemistry and equations are horrible. O well i will just mention a few key words to pick up a couple of marks :dontknow:

Reply 2490

Parthenon93

Original post by Jammers123

I would have thought QT is the length of time taken for ventricular systole :wink:

Okay on that ... So what is "long QT" syndrome? Increased length of ventricular systole so that ventricles do not have enough time to fully relax before another contraction? Ventricles fatigue? Eventually not enough blood supplied to body (brain and heart)? Hence death? ... :eek:

Am I getting this right? :confused:

Reply 2491

Jammers123

Original post by Parthenon93

Am I getting this right? :confused:

This is what I found on wikipedia. The long QT syndrome (LQTS) is a rare inborn heart condition in which delayed repolarization of the heart following a heartbeat increases the risk of episodes of torsade de pointes (TDP, a form of irregular heartbeat that originates from the ventricles). These episodes may lead to palpitations, fainting and sudden death due to ventricular fibrillation.

Reply 2492

claire4095

Original post by Parthenon93

Am I getting this right? :confused:

I think you're right with ventricular fatigue :smile:

if the ventricles fatigue they will be less effective thus lowering the stroke volume and the amount of blood pumped out in each heartbeat? I think, i dont know lol
eeeeek i hate topic 7!

Reply 2493

gildartz

Original post by Parthenon93

Am I getting this right? :confused:

In QT syndrome, the T bit on the ECG is longer, so diastole lasts longer and therefore blood is not pumped out fast enough to meet the demands of the brain. Since the brain is not receiving an adequate supply of blood, you can lose consciousness and leads to "sudden death"

Reply 2494

amnah_70

Original post by claire4095

How are you all revising the scientific article?
Will just reading it over a few times be enough? :/

m using this:
54265374-Questions-and-Answers-for-Scientific-Article-June-2011.pdf

Reply 2495

sheep_go_baa

Just finished the June 2010 paper. Now am proceeding to mark it O_O
*nervousnervousnervous*

Reply 2496

claire4095

Original post by amnah_70

m using this:
54265374-Questions-and-Answers-for-Scientific-Article-June-2011.pdf

Omg thank you thank you thank you!
Does this cover everything they could ask then probably? :smile:

Reply 2497

Maria1234

Explain differences in the mechanisms of substances like IGF-1 and MGF, and transcription factors such as FOXO???????????????????

Reply 2498

amnah_70

Original post by claire4095

Omg thank you thank you thank you!
Does this cover everything they could ask then probably? :smile:

no problem, to be honest lets just hope it does cover evrything as i hate synoptic questions :frown:

Reply 2499

hey-hey-hey

has anyone got 2011 jan snab mark scheme? pleasee

Also on the article, im a bit confused because it says bit on
Catching Cheaters, 4th paragraph, states that:
people with plenty of red blood cells should have little or no eop circulating in the blood.Surely the concentration of eop should be higher?? because they injected the gene for it, so more epo was produced?! to make higher red blood cells, epo is required. How can they have high red blood cells with low epo? shouldnt it be high, then we know if they took it-cheated.
please help me, i dont understand.

(edited 12 years ago)