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The Trp Operon (Attenuation)

I have done some reading, and I understand that the operon is under positive control, so when there is tryptophan it binds to repressor and activates it, allowing it bind to the dna and prevent transcription.

I also understand attenuation, the idea that self complementary regions can form with diff properties, what I don't understand is how it is possible for attenuation to be controlled also by the level of tryptophan, i.e the process only terminates when there is tryptophan trna available, but surely that should activate repressor ?
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