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    Is the tuboglomerular feedback mechanism purported to be inactivated by sustained hyperglycaemia? Why
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    (Original post by Eleanor_mcg)
    Is the tuboglomerular feedback mechanism purported to be inactivated by sustained hyperglycaemia? Why
    I can't think of any logical reason why this might be the case, macula densa cells detect sodium, not glucose. However you could develop a nephropathy from sustained hyperglycaemia,which could prevent the kidneys from controlling fluid balance
 
 
 
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