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Someone please point me in the right direction with attempting this task, any help is much appreciated
Write a discussion summarizing and explaining changes in energy physiology and metabolism of the body to deal with the needs of the body at rest, during and after exercise.
Write a discussion summarizing and explaining changes in energy physiology and metabolism of the body to deal with the needs of the body at rest, during and after exercise.
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Hi, to push you off the cliff [NOT literally! ] look up ALL the following and expand on them:-
AT REST:
1. Heart rate and breathing rate controlled by parasympathetic tone and
modulating influence of cardiac centre and respiratory centre in medulla oblongata
2. Oxygen dissociation curve (sigmoid in shape) ensures base-level saturation with O2 in lungs and release of O2 in tissues.
3. Glycogenesis prevails together with fat deposition - anabolism greater
EXERCISE:
1. increased sympathetic activity -----> Release of adrenaline (alpha + beta effects) and noradrenaline (mainly alpha) ------->
+ve chronotropic effect -----> tahycardia
+ve inotropic effect -------> increased stroke volume
2. Muscle arterioles dilate by action of beta2 adrenoceptors
3. Tachypnoea + deeper ventilation ----- > greater O2 transfer
4. O2 dissociatiion curve shifts to right (Bohr shift) due to rise in CO2 levels -----> more O2 released in tissues
5. More catabolic activity - glycogenolysis prevails
] look up ALL the following and expand on them:-AT REST:
1. Heart rate and breathing rate controlled by parasympathetic tone and
modulating influence of cardiac centre and respiratory centre in medulla oblongata
2. Oxygen dissociation curve (sigmoid in shape) ensures base-level saturation with O2 in lungs and release of O2 in tissues.
3. Glycogenesis prevails together with fat deposition - anabolism greater
EXERCISE:
1. increased sympathetic activity -----> Release of adrenaline (alpha + beta effects) and noradrenaline (mainly alpha) ------->
+ve chronotropic effect -----> tahycardia
+ve inotropic effect -------> increased stroke volume
2. Muscle arterioles dilate by action of beta2 adrenoceptors
3. Tachypnoea + deeper ventilation ----- > greater O2 transfer
4. O2 dissociatiion curve shifts to right (Bohr shift) due to rise in CO2 levels -----> more O2 released in tissues
5. More catabolic activity - glycogenolysis prevails
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(Original post by macpatelgh)
Hi, to push you off the cliff [NOT literally!] look up ALL the following and expand on them:-
AT REST:
1. Heart rate and breathing rate controlled by parasympathetic tone and
modulating influence of cardiac centre and respiratory centre in medulla oblongata
2. Oxygen dissociation curve (sigmoid in shape) ensures base-level saturation with O2 in lungs and release of O2 in tissues.
3. Glycogenesis prevails together with fat deposition - anabolism greater
EXERCISE:
1. increased sympathetic activity -----> Release of adrenaline (alpha + beta effects) and noradrenaline (mainly alpha) ------->
+ve chronotropic effect -----> tahycardia
+ve inotropic effect -------> increased stroke volume
2. Muscle arterioles dilate by action of beta2 adrenoceptors
3. Tachypnoea + deeper ventilation ----- > greater O2 transfer
4. O2 dissociatiion curve shifts to right (Bohr shift) due to rise in CO2 levels -----> more O2 released in tissues
5. More catabolic activity - glycogenolysis prevails
Hi, to push you off the cliff [NOT literally!
] look up ALL the following and expand on them:-AT REST:
1. Heart rate and breathing rate controlled by parasympathetic tone and
modulating influence of cardiac centre and respiratory centre in medulla oblongata
2. Oxygen dissociation curve (sigmoid in shape) ensures base-level saturation with O2 in lungs and release of O2 in tissues.
3. Glycogenesis prevails together with fat deposition - anabolism greater
EXERCISE:
1. increased sympathetic activity -----> Release of adrenaline (alpha + beta effects) and noradrenaline (mainly alpha) ------->
+ve chronotropic effect -----> tahycardia
+ve inotropic effect -------> increased stroke volume
2. Muscle arterioles dilate by action of beta2 adrenoceptors
3. Tachypnoea + deeper ventilation ----- > greater O2 transfer
4. O2 dissociatiion curve shifts to right (Bohr shift) due to rise in CO2 levels -----> more O2 released in tissues
5. More catabolic activity - glycogenolysis prevails
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