Edexcel GCE Biology Unit 4 6BI04 June 2013

Someone has already posted them on this thread..probably on the 3rd or 4th page but m not exactly sure..they are really good.

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I wish.. i feel soo realxed about it.. (ill eat my words late) but i felt this way in jan i felt as if i knew everything and ended up with a C that showed me..but i really dont know what more to do..all the past papers i have done i have got an A* via the UMS but thats probably cause ive done it twice

What are your predictions?
I made a list of all the possible topics and looked at the patterns...
Here is my prediction
-Structure of chloroplast
- Light dependant stage of photosynthesis
-how the products of photosynthesis are used
NPP/GPP
Carbon Cycle
Ecology prac
Niches/Succession
Cause of global warming and the effect of global warming
Evidence for global warming
Nature of the genetic code
Post transcriptional change
DNA profiling
Transcription
Bacteria vs Virus
Barriers to infection
TB
Specific immunity
Antibiotics prac
Hospital Acquired infections

okay basically the HIV virus's GP120 binds onto the CD4 receptors on the membrane of the lymphocyte- T cell, and then enters the cell. Reverse transcriptase turns the RNA into DNA. The cell starts to make viral RNA with help of Intergraze. It will then make viral proteins. Thus new viruses. it will then kill the cell when it leaves the cell. The T killer cells are alerted and they will destroy the T helper cells that are infected. This will lower the overall imunity of the person as there is a reduction in T helper cells. This means that the person is now prone to opertunistic infection. this means they might die from a disease such as TB

what exam board are you doing?
whatever you've mentioned has NOTHING to do with the edexcel syllabus

i think reverse transcriptase converts DNA into Viral RNA isn it?

Thanks
Lytic pathaway is when it leads to cell lysis right? the death of T-helper cells?

yes, the host cells burst = releasing the large number of new virus particles to invade other cells i dont think the lytic pathway has anything to do with t helper cells

Yep
My bad! I was thinking it in terms of HIV - because HIV attacks T-Helpers. Guess its time for me to stop revising

Lol same, I was more prepared in Jan then I am now. I really thought I got an A* in Jan, but I also got a C. I realized it was not the content, that prevented me getting the grade, but it was the exam technique.

The examiners want perfect answers, they want us to be specific. I also noticed in mark schemes, they state the obvious, and that also gets you marks. There is not much we can do really.

Kind of, what I'm doing is going through the specification and checking that I know everything on there. If I don't know it, I'll find past paper questions on that point and go through them, and if I still don't get it, going back to the text book. If you want, I have a grid with all of the specification points on and what past papers they appear in I can upload if you think it will be helpful?

awww no worries im going to stop too, its been a long day, gonna do some in the morning Good Luck

Lol same, I was more prepared in Jan then I am now. I really thought I got an A* in Jan, but I also got a C. I realized it was not the content, that prevented me getting the grade, but it was the exam technique.

The examiners want perfect answers, they want us to be specific. I also noticed in mark schemes, they state the obvious, and that also gets you marks. There is not much we can do really.

Wanna go through some classic answers..and what to put down for each topic so we hit all the marks in the amrk scheme i think that would really help the night before the exam?

Yeah, thanks! The only thing I'm abit uncertain about is for which circumstances would we talk about the cell mediatiated or the humoral?